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GLP-1 RAs exert an antihyperglycemic effect by enhancing pancreatic -cellmediated glucose-dependent insulin secretion, suppressing glucagon secretion, delaying gastric emptying, and reducing food intake in patients with T2D [6]

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Again, the findings backed up the idea that these genetic variants lead to GLP-1 resistance: Those who had the standard PAM gene variants reacted better to GLP-1 medication than those who didn't