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Due to the opposite effects of oncogenes and tumour suppressor genes on cell cycle progression, it was implied that the abnormalities in the growth of tumour cells are a result of the combination of insufficient tumour suppressors (cell cycle breaks) and insufficient oncogenes (cycle accelerators) (28)

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All experiments with mice were performed according to the protocols approved by the Committee of Animal Experiments of Osaka University (approval number 02-001-003), in accordance with the National Institutes of Health Guide for Care and Use of Laboratory Animals

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