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J., Miller, G

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This change weakens inherent neuroprotective mechanisms including mitochondrial biogenesis, synaptic maintenance, and antioxidant defenses in dopaminergic neurons by reducing their ability to remove pathogenic -synuclein and increasing the release of neurotoxins [135]

Therefore, inhibition of mTORC1 pathway and the induction of ISR may conceivably contribute to the pathological conditions related to cysteine deficiency

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