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The HIF switch from acute to chronic hypoxia is mediated by isoform-specific regulators (e.g., HAF/SART1), differential mRNA stability, and ubiquitinproteasome dynamics, all of which contribute to the preferential stabilization and activity of HIF-2 during prolonged hypoxic conditions ( In contrast to HIF-1 and HIF-2, the biological functions of hypoxia-inducible factor-3 (HIF-3) are less well characterized and appear to be more context-dependent (40, 41)

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*Correspondence: Ethan Will Taylor [email protected] This article was submitted to Nutritional Immunology, a section of the journal Frontiers in Nutrition Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Figure 4 MDA, a measure of oxidative stress, was quantified in the plasma, PBMCs, and RBCs isolated from T2DM subjects at pre- and post- L-GSH/or placebo supplementation

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