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Involvement of endoplasmic reticulum Ca2 + release through ryanodine and inositol 1,4,5-triphosphate receptors in the neurotoxic effects induced by the amyloid-beta peptide

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Simultaneously, higher S-adenosylmethionine (SAM) availability supports key methylation-dependent pathways, which are crucial for epigenetic regulation and gene expression related to energy metabolism

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Am J Physiol Endocrinol Metab

Review article references were checked to ensure inclusion of all possibly relevant studies