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However, during inflammatory condition where there is a high demand to consume NADPH, G6PD is released from its inhibition and forms an active homodimer (51, 52)

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The mechanism relevant to tendon healing centers on VEGF pathway modulation and nitric oxide system regulation, both of which support the angiogenic and inflammatory phases of tissue repair

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doi: 10.1186/s12974-022-02564-1 125