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Cleavage at the caspase-6 site is required for neuronal dysfunction and degeneration due to mutant huntingtin

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Importantly, triggering this cascade either via BDNF depletion or C/EBP activation could accelerate A and tau pathology in 3 Tg mice, suggesting that BDNF/TrkB reduction and C/EBP activation may work cooperatively to drive AD pathogenesis

Why it was included in the FDA ban Research has not yet proven MOTS-C to deliver the above benefits