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10.1038/s41598-020-74022-y 29 HsiehM

Galland R et al

& Parrinello, M

Retinoid mechanisms differ fundamentally from GHK-Cu by directly binding nuclear receptors controlling gene expression and cell turnover rather than providing enzymatic cofactors supporting natural processes, creating stronger effects but also more pronounced dependency where discontinuation often triggers rebound effects

311 In addition to the excessively increased consumption of NAD + , multiple viruses cause a decline in NAD + concentration through reducing the protein levels of crucial enzymes in the NAD + biosynthesis pathway, including QPRT and NAMPT

Indian journal of pharmaceutical sciences 73 , 152158 (2011)