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van Hinsbergh VW et al
Kirino Y, Sei M, Kawazoe K, Minakuchi K, Sato Y
Moreover, the mutated core of a GLP-1 peptide was fused to a fragment of a human immunoglobulin also with the intention to prolonged the half-life of the compound and reduce immunogenic side effects (Fig
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In collaboration with researchers in Copenhagen, the team showed that defects in PAM do not affect how GLP-1 binds to its receptor or how signaling occurs at the receptor level
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