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Molecular basis for pore blockade of human Na + channel Nav1.2 by the -conotoxin KIIIA
Previously, we reported that GH-RH acts as a prostatic growth factor in experimental BPH and in autoimmune prostatitis models and its blockade with GH-RH antagonists offers therapeutic approaches for these conditions
Coordination with a healthcare provider helps prevent unintended effects on thyroid function
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