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Animal models indicate that impaired mitochondrial function and glutamate-mediated neurotoxicity may be involved in the pathology of Huntington's disease
Tracking changes beyond body weight can be useful during longer-term treatment
Microarray and RNA-seq data generated in this study have been deposited in the Gene Expression Omnibus under accession code GSE184055 and GSE184261, respectively
doi: 10.1016/S2468-1253(18)30125-0
The results revealed that DBP induced dose-dependent male reproductive dysfunctions, included a significant decrease in the serum testosterone level concomitantly with significant decreases in the sperm count, viability, and total motility
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