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Concurrently, SCFAs enhance intestinal barrier integrity by upregulating tight junction proteins (e.g., occludin), which reduces systemic lipopolysaccharide (LPS) leakage and inhibits TLR4/NF-B-driven synovial inflammation (24, 25)

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However, challenges persist, and further research is necessary to address unmet needs in early detection and screening, develop more effective and personalized therapeutic strategies, and further elucidate the molecular underpinnings of EC