Miro1dependent mitochondrial positioning drives the rescaling of presynaptic Ca2+ signals during homeostatic plasticity
To further explore the role of the BM microenvironment in promoting mTOR activity in LTR, we analyzed mTOR expression in the non-CAR T cell fraction in the BM, which is composed of non-transduced T cells from the CAR T cell product as well as endogenous reconstituting T cells
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Unlike NSAIDs, which blunt inflammation broadly, BPC-157 seems to modulate inflammatory signaling rather than shut it down completely