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This C-terminal metabolite of GLP-1 has shown cytoprotective effects in isolated mouse hepatocytes and cells exposed to oxidative stress injury, as indicated by trafficking to the mitochondria and the limiting of stress-induced apoptosis through inhibition of mitochondrial depolarization, cytochrome c release, and caspase activation (11, 42)

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how the MGBA-regulated astrocytic pathways contribute to AD pathology

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