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Absence of Gclc impairs mitochondrial respiration in FoB Our data above showed that glucose-derived fluxes into the TCA cycle were reduced in GSH-deficient FoB

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Interestingly, a progressive fall of mucosal GSH from 0.5 hours to 4 hours was accompanied by a worsening of injury determined histologically, suggesting that GSH is an important factor in the protection of mucosa against TNBS+ethanol (Fig

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