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The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes
doi: 10.1038/nrd4296 526 CrottyS
It is used to restore vitamin B1, B6 and/or B12 levels
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