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It does not directly stimulate fibroblast proliferation under normal culture conditions, but substantially increases fibroblast survival under hydrogen peroxide-induced oxidative stress acting as a cytoprotective enhancer of the endogenous repair response rather than a direct mitogenic agent
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Wound Healing & Tissue Regeneration By stimulating fibroblast proliferation and vascularization, GHK-Cu accelerates wound closure and scar remodeling
10.7150/ijbs.38487 Summary Keywords redox dysregulation, ALS pathogenesis, oxidative stress, PDIprotein disulfide isomerase, SOD1, ROSreactive oxygen species Citation Jagaraj CJ, Parakh S and Atkin JD (2021) Emerging Evidence Highlighting the Importance of Redox Dysregulation in the Pathogenesis of Amyotrophic Lateral Sclerosis (ALS)
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