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The presence of selenocysteine at the active site of GPx enzymes, which is encoded by the stop codon TGA, is vital for catalytic activity and antioxidant function

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The results showed that the half-maximal inhibitory concentration (IC 50 ) of Amlodipine (AM) against Ishikawa cells (at 72 h) was 12.36 M

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The authors concluded that GHK-Cu accelerates wound healing and skin repair through coordinated effects on collagen and glycosaminoglycan turnover, modulation of metalloproteinase activity, attraction of immune and endothelial cells to injury sites, and restoration of replicative vitality in irradiated fibroblasts [1]

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