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Abstract The c-MET receptor is a receptor tyrosine kinase (RTK) that plays essential roles in normal cell development and motility

The identified proteases responsible for its rapid decay are DPP-4 (dipeptidyl peptidase-4) 34 , which cleaves selectively between residues 2 and 3, and NEP 24.11 35 , which cleaves at multiple sites

It also promotes the downregulation of GPX4 and xCT, ultimately resulting in iron-dependent oxidative cell death

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Structure based development of phenylimidazole-derived inhibitors of indoleamine 2,3-dioxygenase