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Isoquercetin can significantly alleviate retinal damage in mice with DR, and in vitro studies have confirmed that its protective effect is mediated by inhibiting the p53 signaling pathway (227)

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It does so by reducing melanin production through the inhibition of the enzyme tyrosinase, and by converting eumelanin (dark pigment) into pheomelanin (a lighter, reddish-yellow pigment)

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Research suggests that GHS-R activation may influence not only pituitary signaling but also hypothalamic neuropeptide networks involved in appetite regulation and metabolic sensing

A key advantage of these systems lies in their ability to enable personalized therapies