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Glucagon-like peptide 1 signaling inhibits allergen-induced lung IL-33 release and reduces group 2 innate lymphoid cell cytokine production in vivo
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The products of the -oxidation are then used by the Krebs cycle to produce adenosine triphosphate (ATP) as a form of energy [7]
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A recent study explored the mechanisms underlying the functional impairment and metabolic reprogramming of T lymphocytes in multiple myeloma and found silencing SLC38A2 resulted in decreased glutamine uptake and immune dysfunction of T cells [198]
