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Simmons TW et al

In a study, it was shown that Melanotan II was administered to male mice bilaterally into the nucleus accumbens (the brain part involved in rewarding effects of food)

Additionally, tirzepatide was designed to bind to the GIPR with an affinity 5-fold stronger than that for the GLP1R, resulting in greater potency and selectivity in engaging the GIPR over the GLP1R

On the other hand, SWS deficit has been linked to brain abnormalities, and is often reported in cases of schizophrenia and neurodevelopmental disorder [3]