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Astrocyte-specific Bmal1 deletion induces astrocyte activation in vitro and in vivo, mediated in part by suppression of glutathione-s-transferase signaling

The large Nordic cohort had a median follow-up of approximately 2.5 years, and the Chang 2025 study followed patients for 1 year

The article explores alternative molecular mechanismsincluding tau protein aggregation, neuroinflammation, synaptic and mitochondrial dysfunction, brain insulin resistance, vascular impairment, and infectious triggersthat may provide better insights into disease progression and resistance to current treatments

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Curr Opin Organ Transplant