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GLP-1 itself is an incretin hormone, and semaglutide is a structurally modified analog of native GLP-1 built for an extended half-life

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Considering the main causes of the development of T2DM, impaired insulin secretion and insulin resistance, the apparent inefficiency of current pharmaceutical strategies may be due to the undue focus place on alleviating symptoms, such as hyperglycemia by stimulating insulin secretion

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