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Further chain elongation was effected by de-blocking the N-terminus in pentapeptide 9 and coupling of the resulting product with t -butyl N -benzyloxycarbonylaspartate to give hexapeptide 10
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This suppression is independent of plasma cholesterol changes and BW reduction through regulating markers of plaque instability and inflammation including those linked to plaque hemorrhage (CD163), matrix turnover (matrix metalloproteinase (MMP)-3, MMP-13), cholesterol metabolism (prostaglandin I2 synthase (PTGIS), ATP-binding cassette transporter 1 (ABCA1)), leukocyte recruitment (C-C motif chemokine ligand 2 (CCL2), osteopontin (OPN), interleukin (IL)-6), and leukocyte rolling, extravasation and adhesion (vascular cell adhesion molecule (VCAM)-1, E-selectin (SELE)) [16]
The release of inflammatory mediators leads to increased vascular permeability, contributing to inner ear edema and potential disruption of cochlear ion homeostasis
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