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In summary, our data demonstrate that via GIP signalling linagliptin is able to retain its ability to improve glucose tolerance in mice devoid of Gcg -encoded peptides, including GLP-1 and oxyntomodulin (two targets of DPP-4 degradation)

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When combined with semaglutide, which works by slowing stomach emptying and increasing insulin secretion, the two compounds operate through different mechanisms