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In accordance with this observation, we demonstrated that resting wild-type MZB show increased glucose uptake and glycolysis compared to resting FoB

GLP-1R knockout mice (GLP-1R KO, Shanghai Model Organisms, based on C57BL/6N) were generated using CRISPR/Cas9 technology, which causes Glp1r gene protein reading frame shift and loss of function by using non-homologous recombination repair to introduce mutations

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Capsules are convenient but require more pills to reach therapeutic doses

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