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Bioavailability and pharmacokinetics of genistein: mechanistic studies on its ADME

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In malignancy, this constitutes a constitutive, irreversible metabolic adaptation driven by oncogenic signaling (e.g., HIF-1 stabilization, c-Myc amplification) that supports uncontrolled proliferation independent of oxygen availability

The same alkylation shift pattern was observed with strep-MBP-ROXY9- S CLC (Supplementary Fig

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