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GSH, as a cofactor of GPX4, facilitates the reduction of phospholipid hydroperoxides (PLOOHs) to their corresponding alcohols (PLOHs), thereby maintaining redox balance [33]

It may be because substance-use disorders are built upon abnormal glutamate neurotransmission, and that schizophrenia heavily features dopaminergic dysfunction ( 22 )

322 HFD may also enhance neuroinflammation by increasing circulating free fatty acids and cytokines, which may lead to cognitive impairment

The time it takes to repair tissue is also affected by lowered glutathione levels, resulting in more intense and longer durations of soreness, muscle and joint pain, and injuries

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Nemkov, T., Hansen, K