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BPC-157 is created synthetically in labs
We expect in the near future growing recognition of GSH involvement, by S -glutathionylation and/or by the thiols redox state shift, not only in the induction of ATG and/or apoptosis, but also in the modulation of activities of molecular/transcription factors such as NEF2L2, PGC1, and p53 that by inducing the antioxidant defense and assuring mitochondrial homeostasis could block both processes
BPC-157 has shown benefits in animal models of inflammatory bowel disease, NSAID-induced gut damage, and stress ulcers (5)
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Chang SP, Chen YH, Chang WC, Liu IM, Cheng JT
