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This rapid and sustained effect is biologically plausible, as GLP-1 receptors are expressed in key central nervous system areas implicated in reward, motivation, stress, and addiction.20 At clinically therapeutic doses, GLP-1 receptor agonists cross the blood-brain barrier and are thought to modulate dopamine neurotransmission in the mesolimbic system, reducing the rewarding properties of addictive drugs.314202168697071 Other potential mechanisms include the potentiation of inhibitory -aminobutyric acid-ergic tone,72 modulation of the hypothalamic-pituitary-axis, serotonin, and glutamate systems,18192021 and indirect neuroprotective and anti-inflammatory effects that could mitigate the chronic neurobiological consequences of SUDs.62 Limitations and strengths of this study This study has several limitations

The combination approach, reducing both dose and frequency, is the most conservative option and may be best for people who have been on medication for extended periods

Introduction to Semaglutide and GLP-1 Medication Semaglutide and GLP-1 medications have a fascinating history, initially developed as treatments for type 2 diabetes

This case highlights the forensic power of the audit trail

Going forward this provides a valuable yardstick for evaluating the benefits of GLP-1 drugs which have been used since 2005

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