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These similarities with our model carrying TrKB depletion in iSPNs suggest a new concept for BDNFTrkB signalling in the context of striatal protection modulating dopamine circuits through metabolic pathways such as glutathioneascorbate and energy metabolism

Those studies support the broader repair mechanism

Muscat JE, Kleinman W, Colosimo S, Muir A, Lazarus P, Park J et al

doi: 10.3390/nu10030290 70 World Health Organization

One of the major challenges in spinal cord injury is the failure of axons to regrow and reconnect with their target sites (Tohda 2025)

Innovative approaches for cancer treatment: current perspectives and new challenges