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Incubation with GLP-1 and the GLP-1R agonist, exenatide, triggered a cAMP response in MEG-01 cells, with exenatide significantly reducing platelet aggregation induced by thrombin, ADP, and collagen

While the DPP-4 resistance is likely a change to enhance the insulinotropic effect, the changes in the degradation system may have evolved as a countermeasure to the venoms effect mediated by GLP-1 on blood glucose

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They help regulate blood sugar, manage appetite, and often play a significant role in sustainable weight management

This allows for a more complete and balanced healing response