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Therefore, the -1 subcluster was the only cell subcluster to exhibit a consistent increase in IAPP , PCSK1 , and MAFA in response to liraglutide treatment, suggesting that the effect of liraglutide to increase expression of these cell genes was specific to the -1 subcluster

Our models suggest that the reported unimolecular poly-agonist peptide design rules developed in this work have potential applications for further design of potent pharmacologically active peptides in the future to manage metabolic disorders such as diabetes and obesity

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