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Additionally, in animal models, riboflavin deficiency was also found to induce down-regulated expression of key osteogenic markers in the femur, including Runx2, Osterix, and BMP-2/Smad1/5/9 cascade [12]

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Finally, to assess functional relevance, we treated HCD-fed mice with rotenone, a mitochondrial complex I inhibitor, and evaluated survival following CLP-induced sepsis