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Recent insights into the pathogenesis of AA have led to the development of new treatment strategies, such as Janus kinase (JAK) inhibitors, biologics, and several small molecular agents

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IGF-1 LR3, by contrast, works through the classic IGF-1 receptor signaling cascade, activating the PI3K/Akt/mTOR pathway and supporting satellite cells once they have already entered the proliferative and differentiation phase