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Another key limitation is the uncertainty regarding whether CNS penetration of GLP-1 RAs mediates their physiological or therapeutic effects
doi: 10.1002/mnfr.202000426 51 AlshahraniFAljohaniN
Gut (2006) 55:24351
However, Zierke et al., using a Rab7 knockout mouse modelRab7 being a key GTPase involved in lysosomevesicle fusionobserved that trypsinogen activation still occurred and pancreatic injury was not substantially delayed, even when lysosomezymogen granule fusion was disrupted
The valuable role of ethnomedicine in antidiabetic drug development is also exemplified by the history of metformin (1,1-dimethylbiguanide hydrochloride
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