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Previous studies showed that LPS, as a key risk factor for ALI, stimulated alveolar macrophages to activate Toll-like receptor 4 (TLR4) signaling (52), while LPS could also upregulate the expression of chemokines CXCL1, CXCL2 and CCL2 on the margins of macrophages and neutrophils in the lung (53)

That does not mean the peptide caused the problem

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BPC-157 is classified as a gastric pentadecapeptide and is notable for its stability in acidic conditions and high water solubility, properties that are uncommon among peptides of similar size