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These new findings encourage a novel working hypothesis for further research, that a chemical-modification mechanism enables transformation of HGF through interaction or reaction with LASSS under physiological conditions, resulting in the retention of HGFs biological activity after exposure to peroxynitrite, at least until an enhanced form of HGF undergoes metabolic turnover in vivo

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The proposed mechanisms include reducing inflammation, enhancing mitochondrial function, and promoting neuronal survival

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& Simpson, R

Albumin binding as a general strategy for improving the pharmacokinetics of proteins