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This is exemplified by microglia, which promotes injury not only via acute ferroptosis but also through sustained inflammation, a later-phase response that can render a drug administered only at onset ineffective (Liu Z., 2025)

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Thus, in neonatal models, hyperoxia exerts long-term detrimental effects on lung development partly through dysregulated FAO, and therapeutic strategies like L-carnitine supplementation that augment FAO show promise in mitigating hyperoxia-induced lung damage (77)

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The hourly locomotor activity was further analyzed at each five-minute interval (Fig