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Its enzymatic function is based on two aspects: its catalytic activity and the auto- S -glutathionylation of GST by itself on Cys47 and Cys10, both of which disturb the subsequent interaction with c-Jun NH 2 -terminal kinase (JNK), resulting in the formation of a GST multimer 30 (Figure 3)

Furthermore, some researchers speculate that thiamine depletion in the pancreas may be a necessary antecedent for pancreatic inflammation (91)

It should be noted, however, that the majority of these reports remains to be validated in additional patient cohorts and in prospective manner, which explains why none has reached clinical practice yet