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Aging is associated with a decrease in glutathione activity, which can result in increased oxidative stress

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These results suggest that reduced homocysteine causes protein thiol homocysteinylation that may induce smooth muscle cell proliferation by binding to a redox receptor and that may exacerbate atherosclerotic lesion formation by inducing collagen expression (Tyagi, 1998)

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However, it turned out that this medication contained trace amounts of BPC-157

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