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This study showed that doses of 500-1000 mg were sufficient to reduce markers of oxidative stress

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Animal studies and early human research suggest these peptides may speed tendon-to-bone healing, reduce inflammatory cell buildup, and support better pain and function

Garg et al

382 Impaired astrocytic iron homoeostasis triggers secondary neuronal iron toxicity, which together with loss of astrocytic homoeostatic support, triggers neuronal death