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In AD and related tauopathies, hyperphosphorylated tau assembles into neurofibrillary tangles that impair proteasome activity and disrupt cellular homeostasis
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They can be beneficial for patients suffering from Parkinsons disease, Alzheimers disease, and other neurological disorders
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Furthermore, the GLP-1 secretion, which was inhibited by pasireotide in response to a high glucose level with L-glutamine and sodium butyrate stimulation, was significantly lower than the basal GLP-1 secretion in the low-glucose (0.1 mM) state in GLUTag cells (Fig
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