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A possible justification for the modification of BSA/AuNCs was to enhance aurophilic interactions of Au(I)-thiolate complexes on the surface of Au(0) core and rigidify the ligand shell

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Notably, amyloid deposits in ATTR patients correlate with markers of lipid peroxidation, such as 4hydroxytrans2nonenal (HNE) adducts [35] and oxidative DNA damage indicated by 8hydroxy2deoxyguanosine (8OhdG) [34]