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These observations suggest the peptides benefits extend beyond local tissue effects to systemic regulatory improvements
However, secondary preventative interventions such as antiplatelet drugs, -blockers, angiotensin-converting-enzyme (ACE) inhibitors, angiotensin-receptor blockers (ARBs), and HMG-CoA reductase inhibitors (statins) have been shown to significantly reduce CVD risk [3]
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CD38 modulates respiratory syncytial virus-driven proinflammatory processes in human monocyte-derived dendritic cells