Rouar, T., Parik, P., Kucera, O., Barto, M
However, the optic nerve may be uniquely vulnerable to changes in perfusion, autoregulation, metabolic status and local anatomic susceptibility, and favorable systemic vascular effects may not necessarily translate into reduced optic nerve ischemic risk. Beyond their cardiovascular benefits, GLP-1 agents may also influence ocular microvascular physiology
When the breakdown of fatty acids is impaired, they accumulate in the nerve sheath (called myelin)
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Sleep deprivation impairs insulin secretion and glucose tolerance, contributing to T2DM risk [50]
The whole situation becomes a real Catch-22 situation