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One promising strategy is to optimize the GLP-1 RA sequence to achieve a similar agonism effect against the glucose-dependent insulinotropic polypeptide receptor (GIPR)

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and people with conditions that cause nutrient malabsorption and/or gastrointestinal blood loss (such as celiac disease, ulcerative colitis, and Crohn's disease)

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Salusin- participates in high glucose-induced HK-2 cell ferroptosis in a Nrf-2-dependent manner