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Moreover, animal studies have shown that complete inhibition of BACE1 activity results in an elimination of A in neurons, but A is required for certain physiological neuronal functions such as regulation of neuron-glia signaling by enhancing spontaneous astrocyte calcium transient signaling via 7-nAChRs (Lee et al., 3)
Molecular mechanisms underlying Ascl1-mediated astrocyte-to-neuron conversion
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Acute skeletal muscle wasting in critical illness
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