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Results VRK2-deficient PC cells are vulnerable to GSH inhibition To investigate the impact of VRK2 on metabolic reprogramming, we treated VRK2-wild-type (VRK2-WT) and VRK2-knockout (VRK2-KO) CFPAC-1 cell lines with 281 metabolism compounds (Supplementary Table1)

M., Moayyedi, P., & Ford, A

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While there was also conservation in the TM bundle interactions when engaged by the four peptides, differences were identified in how each peptide uses different networks to facilitate G protein coupling

All viral glycoproteins share the characteristic of their assembly into oligomers that necessitates the formation of a disulfide bond and this is inhibited by the reducing action of GSH